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CONCLUSION A growing body of evidence has provided much evidence supporting a role for mTORC1 in sensing diverse environmental conditions and for its ability to link the availability of growth factors and nutrients such as amino acids and glucose to cell growth and proliferation

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14,15,16 Mendelian randomization studies have demonstrated no causal relationship between elevated uric acid levels and the risks of diabetes, coronary heart disease, ischemic stroke, heart failure, body mass index, bone mineral density, coronary artery disease, blood pressure, metabolic syndrome, blood glucose levels, triglyceride levels, diabetes mellitus, serum creatinine levels, glomerular filtration rate, and Parkinson’s disease